To our opinion, nystagmus is a violation of saccadic automaticity.

Unfavorable factors - a mechanic squeeze of structures of saccadic center, cosmic overloads, sea sickness, intoxication (alcoholic, narcotic, toxic) all these break a customary regime of work of saccadic center and as a result pronounced eye movements appear, known in clinic practice as nystagmus.

Consequently, nystagmus as we noted before is a special case of saccadic automaticity (Filin V.A., 1987, 1992, 1997), in conditions determining great asymmetry of eye movement. The causes of asymmetry could be different.

Taking into consideration all said above and despite the traditional point of view it is possible to claim that in the origin of nystagmus a quick phase of sleep is primary while a slow sleep phase is secondary. Nystagmus is the real double-phases oscillation, presented in general by quick eye movements but not by slow movements as L.F. Dell'Osso thinks. In other words, saccades every time take the eye out of equilibrium position and a slow phase of nystagmus each time brings it to the physiological rest i.e. to a zero coordinate area. More details about our concept of nystagmus mechanism one can read in presentation of Filin V.A, and Filin A.V. made in June26-29, 2005 at the Vllth European Neuro-Ophthalmological Conference (EUNOS 2005).