Main page -->
Automaticity of saccades -->
Automaticity of saccades in pathology -->
Mechanism of nystagmus
To our opinion, nystagmus is a violation of saccadic automaticity.
Unfavorable factors - a mechanic squeeze of structures of saccadic center, cosmic
overloads, sea sickness, intoxication (alcoholic, narcotic, toxic) all these break a
customary regime of work of saccadic center and as a result pronounced eye movements
appear, known in clinic practice as nystagmus.
Consequently, nystagmus as we noted before is a special case of saccadic automaticity
(Filin V.A., 1987, 1992, 1997), in conditions determining great asymmetry of eye
movement. The causes of asymmetry could be different.
Taking into consideration all said above and despite the traditional point of view it is
possible to claim that in the origin of nystagmus a quick phase of sleep is primary while
a slow sleep phase is secondary. Nystagmus is the real double-phases oscillation, presented
in general by quick eye movements but not by slow movements as L.F. Dell'Osso thinks.
In other words, saccades every time take the eye out of equilibrium position and a slow
phase of nystagmus each time brings it to the physiological rest i.e. to a zero coordinate
area. More details about our concept of nystagmus mechanism one can read in
of Filin V.A, and Filin A.V. made in June26-29, 2005 at the Vllth European
Neuro-Ophthalmological Conference (EUNOS 2005).